Alzheimer’s disease, recognized as a distinct condition by Alois Alzheimer in the early 1900s, is a progressive neurodegenerative disorder and discovery of amyloid beta (Aβ) plaques in the brain has driven research into Aβ aggregation, with metal ions like Cu2+ playing a key role in this process. In the last few decades, compounds like Semax which developed as a neuroprotective agent, has shown the ability to block Aβ
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
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Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.