Alzheimer's disease, first time was outlined by Alois Alzheimer (German psychiatrist) in 1906, this type of dementia is characterized by the buildup of amyloid beta (Aβ) protein in the brain. Despite the exploration of many compounds, no treatment has succeeded. Semax which is a heptapeptide known for its ability to protect humans brain and has favorable potential, in preventing the effects of Aβ accumulation. Research in recent decades has uncovered the role of metal ions, particularly copper (Cu2+), in modulating Aβ aggregation, contributing to the formation of toxic plaques.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
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Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
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