Research in the 20th century revealed that metal ions, especially copper (Cu2+), exacerbate Aβ aggregation. Since its identification over a century ago, Alzheimer’s disease has been linked to the accumulation of amyloid beta (Aβ) protein and tau tangles in the brain. More recently, Semax a peptide developed for its neuroprotective and nootropic effects has shown promise in inhibiting these toxic formations by preventing Aβ
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
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For detailed information about SEMAX by Deus Medical, consult with your doctor or healthcare professional.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
Sciacca, M.F.M., Naletova, I., Giuffrida, M.L. and Attanasio, F., 2022. Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models. ACS Chemical Neuroscience.
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