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Dutasteride

Dutasteride

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Substance Overview and History

In the 1990s GSK developed Dutasteride as a type dual 5 alpha reductase inhibitor that gained FDA approval in 2001. It functions by blocking both the type 1 and type 2 isoforms, of the enzyme to halt testosterone from being transformed into dihydrotestosterone ( DHT).This attribute is especially beneficial in addressing conditions like prostate hyperplasia ( BPH ) and male pattern baldness (androgenic alopecia). Dutasterides scope extends beyond finasteride as it addresses DHT production in tissues such, as the scalp,liver and prostate​​.

References:

Llewellyn, W. (2017). William Llewellyn's Anabolics.
United States: Molecular Nutrition, LLC.

Disclaimer: Information provided it this page is for general information only and does not substitute for professional medical advice.

For more details consult with your doctor or healthcare professional.

FAQ
How is Dutasteride working?
Dutasteride blocks the conversion of testosterone to DHT by inhibiting both type-1 and type-2 5-alpha reductase.
References:

Llewellyn, W. (2017). William Llewellyn's Anabolics.
United States: Molecular Nutrition, LLC.

What makes Dutasteride more effective compare to Finasteride?
Dutasteride inhibits both type-1 and type-2 reductase, while finasteride targets only type-2, making it more effective in reducing DHT.
References:

Llewellyn, W. (2017). William Llewellyn's Anabolics.
United States: Molecular Nutrition, LLC.

When was Dutasteride first approved?
Dutasteride was FDA-approved in 2001​.
References:

Llewellyn, W. (2017). William Llewellyn's Anabolics.
United States: Molecular Nutrition, LLC.